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Daniela COTA

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Médecine, Univ. Bologne, Italie (1999)
Postdoc Institut Max-Planck, Munich (2001–2003)
Postdoc Univ. Cincinnati, USA (2004-2007)
CR1 à l'Inserm (2008-2018)
DR2 à l'Inserm (2018)

Oct 1999: Degree in Medicine and Surgery (M.D., Magna cum Laude), University of Bologna, Italy
May 2000: Medical license


Since 2018: DR2 INSERM

Since 2008: Team Leader, Team: “Régulation de l'équilibre énergétique et obésité” (physiopathology of energy balance and obesity), NeuroCentre Magendie, Bordeaux, France

2008-2018: CR1 INSERM

2004– 2007: Postdoctoral Fellow with Profs. R. J. Seeley and S. C. Woods, Obesity Research Center, University of Cincinnati, USA

2001–2003: Postdoctoral Fellow with Profs. G. K. Stalla and U. Pagotto, Clinical Neuroendocrinology Group, Max-Planck institute of Psychiatry, Munich, Germany

2001–2003: Medical School of Specialization in Endocrinology and Metabolic Disorders, Director Prof. Renato Pasquali, University of Bologna, Italy


73 publication(s) depuis Juin 2000:

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Les IF indiqués ont été collectés par le Web of Sciences en

12/2001 | Eat Weight Disord-st   IF 3.6
Relationship between socio-economic and cultural status, psychological factors and body fat distribution in middle-aged women living in Northern Italy.
Cota D, Vicennati V, Ceroni L, Morselli-Labate AM, Pasquali R.


11/2000 | Recenti Prog Med   IF 0.3
[Steroid therapy and adrenal function].
Cota D, Ceroni L, Pasquali R

Glucocorticoids are frequently used for both diagnostic and therapeutic purposes. Their action mimics endogenous glucocorticoid actions by altering the activity of the hypothalamic-pituitary-adrenal (HPA) axis. Therefore, they can be responsible for iatrogenic diseases, particularly if used at high doses and for a long time. The aim of this brief review is to show the main pharmacological characteristics and the endocrine effects of glucocorticoids. The HPA axis insufficiency, related to acute glucocorticoid withdrawal, is also discussed.

06/2000 | Minerva Endocrinol   IF 1.5
[Pseudo-Cushing syndrome. Physiopathologic aspects and differential diagnosis].
Ceroni L, Cota D, Pasquali R

Pseudo-Cushing Syndromes (PCS) are a heterogeneous group of disorders, including alcoholism and depression, that share many of the clinical and biochemical features of Cushing's Syndrome (CS). It has been suggested that hypercortisolism of PCS may be the result of increased hypothalamic corticotropin-releasing hormone secretion in the context of a hypothalamic-pituitary-adrenal axis that is otherwise normally constituted. The substantial overlap in clinical features and daily urinary free cortisol levels between several patients with CS and those with PCS can make the differential diagnosis difficult. The most accurate tests in the distinction of CS from alcohol-induced PCS are dexamethasone-CRH and a midnight serum cortisol measurement. In depressed patients, the insulin tolerance test may be useful, although some overlap may exist. This brief review summarises the principal pathophysiological events of PCS and provides a useful strategy for differential diagnosis.